Second Episodes of Fits

Sudah lama mama tidak menulis di blog ini.Banyak kekalutan yang berlaku tidak disangka-sangka.Pada tanggal 28 Feb 2012, Sofea diserang oleh Mr. S (sawan) selam lebih kurang 30 min (continuously jerking).

WHY SECOND EPISODES???

Serangan ini dikategorikan sebagai serangan kali kedua kerana semasa umur Sofea 1 hari, Sofea telah diserang sawan selama 1 jam 3+ min di NICU Hospital Sxxxxxx (Punca: Perinatal Asphyxia with hypoxic-ischemic encephalopathy (HIE) Stage III.

Kejadian sawan kali ini berlaku pada jam 6:26 pm.Imbas kembali sebelum kejadian diserang Mr. S (untuk berkongsi pengalaman)

25 Feb 2012, 9:30am: Sofea menjalani sesi fisioterapi di Sham Therapy Centre
                      7:00pm: Kami pulang ke Ipoh
                      10:oopm: Kami tiba di Ipoh

26 Feb 2012, 5:30pm: Sofea ikut mama keluar ke Shop Lot (Naik atas utk lihat papa pasang perabot)
                                    tetapi sekejap sahaja sebab berhabuk  dalam 10 min
                      7:00pm:Kami ke Sungai Siput
                      10:00pm:Kami Tiba semula ke Meru Heights

27 Feb 2012, 6:00pm: Kami tiba di Kuala Lumpur

28 Feb 2012, 8:30am: Sofea bangun seperti selalu
                      9:00am:Sofea minum susu
                      10:30am: Sofea tidur
                       11:00am: Sofea Mandi
                       12:00noon: Sofea minum Susu
                       12:30pm: Sofea tidur semula
                         02:00pm: Sofea bgn tidur
                         02:15pm-02:50pm: Sofea main piano, sensory (soy bean & barley)
                         03:30pm: Sofea minum susu
(Sofea menguap,merengek, dan ada aksi terperanjat (startle) jadi minum susu meleret sampai dekat pukul 5pm)

                        05:30pm: Sofea ada batuk-batuk kecil, tidak bermaya, menguap dan aksi terperanjat
                                      dtg semula
                        05:30pm: Disebabkan Sofea tidak bermaya (mama fikir mungkin kepenatan
                                        balik kampung, Sofea tidak demam)mama hanya berbual-bual dengan Sofea,
                                        mama fikir mungkin esok baru buat intensif fisio utk Sofea, sbb Sofea penat
                        06:oopm: Sofea terperanjat lagi
                        06:15pm: Sofea terperanjat lagi
                        06:26pm: Sofea terperanjat dan selang beberapa minit kemudian Sofea terus
                                       "jerking"dan tidak berhenti, Sofea mengeluarkan bunyi, kaki, tangan, dan
                                        otot muka disebelah kanan "bergetar" tetapi mata Sofea tidak pandang ke atas,
                                        air liur mengalir tetapi mulut tidak berbuih.

 (mama telefon papa, tidak tahu apa yang harus dilakukan, mama hanya lap kepala Sofea dengan air sbb   mungkin Sofea panas, tetapi tetap tidak berkesan, sebelum pergi ke hospital mama "check" pampers Sofea tidak terbuang air besar), semasa dlm kereta ketika dalam perjalanan ke hospital, Sofea tidak lagi mengalami "Jerking" tetapi, wajahnya membiru dan dia dlm keadaan sangat sejuk)

                        06:55pm:kami tiba di hospital kerana jalan sesak

***Begitulah kejadian yang berlaku tempoh hari, mama agak kesal berkenaan hal "terperanjat" Sofea, sebelum ini mama ada bertanyakan tentang perihal "terperanjat" Sofea dengan doktor (specialist) yang merawatnya di NICU Hospital Sxxxxxx, tetapi beliau menafikan itu adalah sawan.***beliau menyatakan bahawa "kanak-kanak seperti Sofea otak mereka akan menghasilkan "electricity", yang menyebabkan kanak-kanak seperti Sofea suka "terperanjat". Tiada ubat dibekalkan untuk masalah "terperanjat" Sofea***Untuk pengetahuan pembaca tiada sebarang ujian MRI, EEG sepanjang Sofea berada di NICU Hospital Sxxxxxx.

Sekurang-kurangnya sekiranya kami tahu lebih awal, mungkin kami dapat bertindak seperti yang sepatutnya :-C

Setelah kejadian ini, doktor menyatakan bahawa Sofea mengalami epilepsy
 (di sini saya ingin berkongsi maklumat berkenaan epilepsy)

Source: http://en.wikipedia.org/wiki/Epilepsy
Epilepsy is usually controlled, but not cured, with medication. However, over 30% of people with epilepsy do not have seizure control even with the best available medications. Surgery may be considered in difficult cases.^[8][9] Not all epilepsy syndromes are lifelong – some forms are confined to particular stages of childhood. Epilepsy should not be understood as a single disorder, but rather as syndromic with vastly divergent symptoms, all involving episodic abnormal electrical activity in the brain and numerous seizures. 

Causes

The diagnosis of epilepsy usually requires that the seizures occur spontaneously. Nevertheless, certain epilepsy syndromes require particular precipitants or triggers for seizures to occur. These are termed reflex epilepsy. For example, patients with primary reading epilepsy have seizures triggered by reading^{[citation needed]}. Photosensitive epilepsy can be limited to seizures triggered by flashing lights. Other precipitants can trigger an epileptic seizure in patients who otherwise would be susceptible to spontaneous seizures. For example, children with childhood absence epilepsy may be susceptible to hyperventilation. In fact, flashing lights and hyperventilation are activating procedures used in clinical EEG to help trigger seizures to aid diagnosis. Finally, other precipitants can facilitate, rather than obligately trigger, seizures in susceptible individuals. Emotional stress, sleep deprivation, sleep itself, heat stress, alcohol and febrile illness are examples of precipitants cited by patients with epilepsy. Notably, the influence of various precipitants varies with the epilepsy syndrome.^[11] Likewise, the menstrual cycle in women with epilepsy can influence patterns of seizure recurrence. Catamenial epilepsy is the term denoting seizures linked to the menstrual cycle.^[12]

There are different causes of epilepsy that are common in certain age groups.

• During the neonatal period and early infancy the most common causes include hypoxic-ischemic encephalopathy, CNS infections, trauma, congenital CNS abnormalities, and metabolic disorders.
• During late infancy and early childhood, febrile seizures are fairly common. These may be caused by many different things, some thought to be things such as CNS infections and trauma.
• During childhood, well-defined epilepsy syndromes are generally seen.
• During adolescence and adulthood, the causes are more likely to be secondary to any CNS lesion. Further, idiopathic epilepsy is less common. Other causes associated with these age groups are stress, trauma, CNS infections, brain tumors, illicit drug use and alcohol withdrawal.
• In older adults, cerebrovascular disease is a very common cause. Other causes are CNS tumors, head trauma, and other degenerative diseases that are common in the older age group, such asdementia.^[13]

When investigating the causes of seizures, it is important to understand physiological conditions that may predispose the individual to a seizure occurrence. Several clinical and experimental data have implicated the failure of blood–brain barrier (BBB) function in triggering chronic or acute seizures,^[14][15] some studies implicate the interactions between a common blood protein—albumin andastrocytes.^[16] These findings suggest that acute seizures are a predictable consequence of disruption of the BBB by either artificial or inflammatory mechanisms. In addition, expression of drug resistance molecules and transporters at the BBB are a significant mechanism of resistance to commonly used anti-epileptic drugs.<sup>[17

Wallahualam...Semoga Sofea dijauhkan daripada serangan Epilepsy lagi....</sup>